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Correction: PSF functions as a repressor of hypoxia-induced angiogenesis by promoting mitochondrial function

The Original Article was published on 11 February 2021

Correction: Cell Commun Signal 19, 14 (2021)

https://doi.org/10.1186/s12964-020-00684-w

Following publication of the original article [1], the authors reported an error in Fig. 5b and c.

Due to an error in the manuscript preparation and proofreading stage, there are mistakes in Fig. 5b and c, including image misuse (Fig. 5b) and incorrect labeling (Fig. 5b and c). The figure presented in this correction article has been corrected.

Fig. 5
figure 1

b-c PSF-HIF-1α complex formation under hypoxia induction. Ectopically expressed PSF interacted with HIF-1α were checked using anti-HA antibody in HRMECs as indicated (a) and anti-His antibody sedimentation (b). c Anti-HIF-1αor anti-PSF antibody-based immunoprecipitation with total cell lysates of HRMECs under hypoxia exposure was performed, followed by immunoblotting with the corresponding antibodies as indicated

Reference

  1. Dong L, Li W, Lin T, et al. PSF functions as a repressor of hypoxia-induced angiogenesis by promoting mitochondrial function. Cell Commun Signal. 2021;19:14. https://doi.org/10.1186/s12964-020-00684-w.

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Correspondence to Lijie Dong or Xiaorong Li.

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Dong, L., Li, W., Lin, T. et al. Correction: PSF functions as a repressor of hypoxia-induced angiogenesis by promoting mitochondrial function. Cell Commun Signal 21, 183 (2023). https://doi.org/10.1186/s12964-023-01237-7

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  • DOI: https://doi.org/10.1186/s12964-023-01237-7