Skip to main content

Correction: MicroRNA-644a promotes apoptosis of hepatocellular carcinoma cells by downregulating the expression of heat shock factor 1

The Original Article was published on 14 June 2018

Correction to: Cell Communication and Signaling (2018) 16:30 https://doi.org/10.1186/s12964-018-0244

Following publication of the original article [1], the authors reported errors that were inadvertently introduced in Figs. 5 and 6. Specifically, the icons of b and c in Figs. 5 and 6, as well as the flow cytometry analysis of HepG2 and SMMC-7721 cells transfected with HSF1-siRNA in Fig. 5 and the SMMC-7721 cells transfected with miR-644a+ HSF1 mut. In Fig. 6 are incorrect in the original publication. The authors sincerely apologize for the error and confirm that this correction does not change the conclusion of the article. The corrected images has been replaced as follows:

Fig. 5
figure 5

HSF1 silencing decreases proliferation and increases apoptosis of HCC cells. a CCK8 assay shows proliferation of HepG2 and SMMC-7721 cells transfected with control or HSF1-siRNA. b Colony formation assay shows the total number of colonies formed by HepG2 and SMMC-7721 cells transfected with control or HSF1-siRNA. c Flow cytometry analysis shows apoptosis of HepG2 and SMMC-7721 cells transfected with control or HSF1-siRNA. Note: * denotes P < 0.05 and ** denotes P < 0.01 compared to controls

Fig. 6
figure 6

HSF1 overexpression promotes proliferation and inhibits apoptosis of HCC cells. a CCK8 assay shows proliferation of HepG2 and SMMC-7721 cells co-transfected with miR-644a mimic and wild-type or mutant HSF1 (wild type or mutant 3’UTR). b Colony formation assay shows the total number of colonies in HepG2 and SMMC-7721 cells co-transfected with miR-644a mimic and wild-type or mutant HSF1. c Flow cytometry analysis of percent apoptosis (AnnexinV+ PI+) in HepG2 and SMMC-7721 cells co-transfected with miR-644a mimic and wild-type or mutant HSF1. Note: * denotes P < 0.05 and ** denotes P < 0.01 compared to controls

Reference

  1. Liang W, Liao Y, Li Z, et al. MicroRNA-644a promotes apoptosis of hepatocellular carcinoma cells by downregulating the expression of heat shock factor 1. Cell Commun Signal. 2018;16:30. https://doi.org/10.1186/s12964-018-0244-z.

    Article  CAS  PubMed  PubMed Central  Google Scholar 

Download references

Author information

Authors and Affiliations

Authors

Corresponding authors

Correspondence to Bo Tang or Zhenran Wang.

Additional information

Publisher's Note

Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.

Rights and permissions

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

Reprints and permissions

About this article

Check for updates. Verify currency and authenticity via CrossMark

Cite this article

Liang, W., Liao, Y., Li, Z. et al. Correction: MicroRNA-644a promotes apoptosis of hepatocellular carcinoma cells by downregulating the expression of heat shock factor 1. Cell Commun Signal 20, 151 (2022). https://doi.org/10.1186/s12964-022-00975-4

Download citation

  • Published:

  • DOI: https://doi.org/10.1186/s12964-022-00975-4