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Correction: Cholecystectomy promotes colon carcinogenesis by activating the Wnt signaling pathway by increasing the deoxycholic acid level

The Original Article was published on 25 May 2022

Correction to: Cell Communication and Signaling (2022) 20:71 https://doi.org/10.1186/s12964-022-00890-8

Following publication of the original article [1], the authors identified an error in Fig. 6E. The updated Fig. 6 is given in this correction article. The original article [1] has been corrected.

Fig. 6
figure 6

GW4064 and GSK inhibitor rescue the promoting effect and inhibiting effect of DCA and GW4064 by regulating the cytosolic β-catenin expression in CC line cells. a, b Living-cell image was used to detect the proliferate capability of CC cells treated with DCA and DCA + GW4064. c, d Living-cell image was used to detect the proliferate capability of CC cells treated with GW4064 and GW4064 + GSK inhibitor. e Western blotting was used to detect the protein level of cytosolic β-catenin in CC cells of NC, DCA, DCA + GW4064. f Western blotting was used to detect the protein level of cytosolic β-catenin in CC cells of DMSO, GW4064, and GW4064 + GSK inhibitor. *P < 0:05, **P < 0.01, ***P < 0.001

Reference

  1. Yao Y, Li X, Xu B, et al. Cholecystectomy promotes colon carcinogenesis by activating the Wnt signaling pathway by increasing the deoxycholic acid level. Cell Commun Signal. 2022;20:71. https://doi.org/10.1186/s12964-022-00890-8.

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Correspondence to Lan Sun, Zheng Zhang or Peng Li.

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Yao, Y., Li, X., Xu, B. et al. Correction: Cholecystectomy promotes colon carcinogenesis by activating the Wnt signaling pathway by increasing the deoxycholic acid level. Cell Commun Signal 20, 85 (2022). https://doi.org/10.1186/s12964-022-00908-1

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  • DOI: https://doi.org/10.1186/s12964-022-00908-1