Fig. 7From: The scaffold of neutrophil extracellular traps promotes CCA progression and modulates angiogenesis via ITGAV/NFκBSchematics highlighting the major findings of this study. Increased NETs deposition in tumor microenvironment was responsible for CCA proliferation, metastasis and angiogenesis, which was driven predominantly by interaction with αV integrin and activation of the NFκB pathwayBack to article page