Fig. 2

Effects of prenatal dexamethasone exposure (PDE) on glucose and lipid metabolism in the adult male offspring rats at postnatal week 28. A Serum glucose level (n = 8); (B) Serum triglyceride (TG) level (n = 8); (C) Serum total cholesterol (TCH) level (n = 8); (D) Serum high-density lipoprotein-cholesterol (HDL-C) level (n = 8); (E) Serum low-density lipoprotein-cholesterol level (n = 8); (F) The ratio of TG to HDL-C levels (n = 8); (G) The ratio of TCH to HDL-C levels (n = 8); (H) The ratio of LDL-C to HDL-C levels (n = 8); (I) Histological analysis of multiple organs (Scale bar = 500 μm); (J) Serum insulin level (n = 8); (K) Insulin resistance index (IRI) (n = 8); (L) Intraperitoneal glucose tolerance test (IPGTT) and the area under curve (AUC) (n = 8); M. Insulin tolerance test (ITT) and AUC (n = 8). Mean ± S.E.M. ^* P < 0.05, ^** P < 0.01 vs. the control group. NS, no significance; PDE(L), prenatal dexamethasone exposure at a low dose (0.2 mg/kg∙d)