Fig. 5From: AIP1 suppresses neovascularization by inhibiting the NOX4-induced NLRP3/NLRP6 imbalance in a murine corneal alkali burn modelSchematic diagram of the proposed mechanism. The protective effect of the NOX4 inhibitor and AIP1 on corneal neovascularization after alkali burn injury is associated with a reduction in ROS production and alleviation of the imbalance in NLRP3 activation and NLRP6 suppressionBack to article page