Skip to main content
Fig. 4 | Cell Communication and Signaling

Fig. 4

From: Endothelial colony-forming cell-derived exosomal miR-21-5p regulates autophagic flux to promote vascular endothelial repair by inhibiting SIPL1A2 in atherosclerosis

Fig. 4

ECFC-derived exosomes improve the proliferation, migration, and tube formation of ox-LDL-treated HMECs in an autophagic flux-dependent manner. A The viability of HMECs with different treatments was measured by CCK-8 assay. Biological replicates = 5, and technical replicates = 3. B, E Cell migration of HMECs with different treatments was determined by wound healing assay. Scale bars = 100 μm. Biological replicates = 4, and technical replicates = 1. C, F Tube formation ability of HMECs with different treatments was evaluated by tube formation assay. Scale bars = 100 μm. Exos ECFCs-exosomes. Biological replicates = 3 and technical replicates = 1. Significant differences between different treatment groups are indicated as *P < 0.05 and **P < 0.01

Back to article page

Cell Communication and Signaling

ISSN: 1478-811X