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Fig. 4 | Cell Communication and Signaling

Fig. 4

From: Tobacco smoke condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-κB and MAPKs P38 and ERK pathways activation

Fig. 4

Colchicine inhibited NF-κB activation in tobacco smoke-condensate-treated endothelial cells. (A) WB showing protein expression and phosphorylation of NF-κB subunit P65. (B) The quantification of the relative protein expression of NF-κB subunit P65 and (C) phosphorylated NF-κB subunit P65. (D) The ratio of the relative protein expression of phosphorylated NF-κB subunit P65 and the relative protein expression of NF-κB subunit P65. The experiments were performed with biological triplicates. β-actin was used as a control. *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001

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Cell Communication and Signaling

ISSN: 1478-811X