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Fig. 1 | Cell Communication and Signaling

Fig. 1

From: Tobacco smoke condensate-induced senescence in endothelial cells was ameliorated by colchicine treatment via suppression of NF-κB and MAPKs P38 and ERK pathways activation

Fig. 1

Colchicine prevented oxidative stress and oxidative stress-induced DNA damage in endothelial cells treated with smoke condensate. (A) DCFH-DA staining and (B) quantification of DCFH-DA staining. (C) Immunofluorescence (IF) staining for 8-OHDG and (D) quantification of 8-OHDG. (E) WB showing protein expression of KU70/KU80. Quantification of relative protein expression of (F) KU70 and (G) KU80. All experiments were performed in triplicates. β-actin was used as a control. SC = tobacco smoke condensate, *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001, scale bar = 100 μm

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Cell Communication and Signaling

ISSN: 1478-811X