Fig. 8From: Inhibition of protein tyrosine phosphatase improves mitochondrial bioenergetics and dynamics, reduces oxidative stress, and enhances adipogenic differentiation potential in metabolically impaired progenitor stem cellsAnalysis of mitochondria metabolism during adipogenic differentiation. Using RT-qPCR, we analysed expression of genes related to mitoribosomes and mitochondrial oxidative phosphorylation (OXPHOS) system-NDUFA9 (a), UQCRC2 (b), COX4I1 (c), COX4I2 (d), MRPL9 (e), MTERF4 (f), OX1AL (g), PPARGC1B (h), PUSL1 (i), BIGBOS (j), MIEF1-MP (k) and TFAM (l) in cells cultured in adipogenic conditions. Results are expressed as mean ± SD; n = 3. ∗p < 0.05, ∗∗p < 0.01, and ∗∗∗p < 0.001. Statistical significance indicated as asterisk (*) when comparing the result to ASCs HE and as number sign (#) when compared to ASCs EMSBack to article page