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Fig. 8 | Cell Communication and Signaling

Fig. 8

From: Inhibition of protein tyrosine phosphatase improves mitochondrial bioenergetics and dynamics, reduces oxidative stress, and enhances adipogenic differentiation potential in metabolically impaired progenitor stem cells

Fig. 8Fig. 8

Analysis of mitochondria metabolism during adipogenic differentiation. Using RT-qPCR, we analysed expression of genes related to mitoribosomes and mitochondrial oxidative phosphorylation (OXPHOS) system-NDUFA9 (a), UQCRC2 (b), COX4I1 (c), COX4I2 (d), MRPL9 (e), MTERF4 (f), OX1AL (g), PPARGC1B (h), PUSL1 (i), BIGBOS (j), MIEF1-MP (k) and TFAM (l) in cells cultured in adipogenic conditions. Results are expressed as mean ± SD; n = 3. ∗p < 0.05, ∗∗p < 0.01, and ∗∗∗p < 0.001. Statistical significance indicated as asterisk (*) when comparing the result to ASCs HE and as number sign (#) when compared to ASCs EMS

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