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Fig. 1 | Cell Communication and Signaling

Fig. 1

From: Inhibition of protein tyrosine phosphatase improves mitochondrial bioenergetics and dynamics, reduces oxidative stress, and enhances adipogenic differentiation potential in metabolically impaired progenitor stem cells

Fig. 1Fig. 1

Analysis of mitochondria metabolism in undifferentiated ASCs. Expression of NDUFA9 (a), UQCRC2 (b), COX4I1 (c), COX4I2 (d), MRPL24 (e), MTERF4 (f), OX1AL (g), PPARGC1B (h), PUSL1 (i), BIGBOS (j), MIEF1-MP (k) and TFAM (l) was investigated with RT-qPCR. Results are expressed as mean ± SD; n = 3.∗p < 0.05, ∗∗p < 0.01, and ∗∗∗p < 0.001

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