Fig. 9From: TRAF1 suppresses antifungal immunity through CXCL1-mediated neutrophil recruitment during Candida albicans intradermal infectionModel of TRAF1-mediated antifungal immunity in the skin. Schematic presentation of the role of TRAF1 during C. albicans intradermal infection. TRAF1-deficiency upregulates CXCL1 production and neutrophil recruitment through increasing the activation of NFκB in the macrophages or decreasing STAT1 activation in the keratinocytes, respectively, leading to a better clearance of fungi in the skinBack to article page